April 3, 2020: "A collaborative study led by the Monash Biomedicine Discovery Institute (BDI)... has shown that an anti-parasitic drug (IVM) kills the virus within 48 hours."
From the World Ivermectin Day website
Dear STEMs and NAVs.
Please dissect zeeee following Ivermectin science and the claims
https://worldivermectinday.org/monash-university-shows-ivermectin-eliminated-sars-cov-2/
From the World Economic err….Ivermectin Day timeline, (which does not go back decades to when Merck and Bill Gates began shipping it for free to the African nations, but picks up during the Covid Psy Op of FDA Horsie Meds Bad.)
Okay.
Right quick in the World Economic Ivermectin Day timeline we got to a study!
April 3, 2020
Ivermectin SARS-Cov-2 Study
A collaborative study led by the Monash Biomedicine Discovery Institute (BDI) with the Peter Doherty Institute of Infection and Immunity (Doherty Institute), a joint venture of the University of Melbourne and Royal Melbourne Hospital, has shown that an anti-parasitic drug already available around the world kills the virus within 48 hours.
The Monash Biomedicine Discovery Institute’s Dr Kylie Wagstaff, who led the study, said the scientists showed that the drug, Ivermectin, stopped the SARS-CoV-2 virus growing in cell culture within 48 hours.
“We found that even a single dose could essentially remove all viral RNA by 48 hours and that even at 24 hours there was a really significant reduction in it.”
Dr Wagstaff
Ivermectin is an FDA-approved anti-parasitic drug that has also been shown to be effective in vitro against a broad range of viruses including HIV, Dengue, Influenza and Zika virus.
Dr Wagstaff cautioned that the tests conducted in the study were in vitro and that trials needed to be carried out in people.
“Ivermectin is very widely used and seen as a safe drug. We need to figure out now whether the dosage you can use it at in humans will be effective – that’s the next step,” Dr Wagstaff said.“In times when we’re having a global pandemic and there isn’t an approved treatment, if we had a compound that was already available around the world then that might help people sooner. Realistically it’s going to be a while before a vaccine is broadly available.
Although the mechanism by which Ivermectin works on the virus is not known, it is likely, based on its action in other viruses, that it works to stop the virus ‘dampening down’ the host cells’ ability to clear it, Dr Wagstaff said.
Royal Melbourne Hospital’s Dr Leon Caly, a Senior Medical Scientist at the Victorian Infectious Diseases Reference Laboratory (VIDRL) at the Doherty Institute where the experiments with live coronavirus were conducted, is the study’s first author.
“As the virologist who was part of the team who were first to isolate and share SARS-COV2 outside of China in January 2020, I am excited about the prospect of Ivermectin being used as a potential drug against COVID-19,” Dr Caly said.
My immediate thoughts and questions.
This is going to be very long. Warning.
an anti-parasitic drug already available around the world kills the virus within 48 hours.
The Monash Biomedicine Discovery Institute’s Dr Kylie Wagstaff, who led the study, said the scientists showed that the drug, Ivermectin, stopped the SARS-CoV-2 virus growing in cell culture within 48 hours.
SARS-Cov-2 growing in cell culture.
SARS-Cov-2 growing in cell culture.
SARS-Cov-2 growing in cell culture.
Does this refer to a cell culture created somehow from genetic materials on hand and then fabricated by pulling from the Chinese computer sequence emailed around the world by China?
Is this what JJ Couey is talking about with infectious clones?
Brewing a sequence up in like e.coli or some shit…literally?
Or did they find some poor bastard with a cough and have him hack up some sputum and presto…start GROWING THE SARS-COV-2!
“We found that even a single dose could essentially remove all viral RNA by 48 hours and that even at 24 hours there was a really significant reduction in it.”
Again, can you show your work?
Has this been duplicated?
The root question is where did the viral RNA come from for the purposes of the study?
Next question: Steve Kirsch and Billionaire Jeffrey Skoll, maybe you guys could use some of that Philanthropy dough and run this study on the VIRAL RNA, you know that is…SARS-COV2. 👍
Of course, Kirsch had his own pet cure for the SARS-Cov-2 super virus which spread the world and was murdering everybody.
Anthony Colpo writes:
Hi Steve,
you've done a lot of good work in calling out the toxic COVID 'vaccines', so it's terribly sad to see you glorifying a truly garbage drug like fluvoxamine.
Your article ignores several key points:
1. The Thai study you cite in this article is not worth the paper it's written on. It's a completely unblinded, open-label study, meaning the researchers knew which group each subject was in. When one of those researchers, Angela M Reiersen, just happens to hold a patent for the use of fluvoxamine as a COVID treatment, that's a problem. A big problem. The potential for bias and misreporting of the results is obvious.
2. There clearly was misreporting in this study, because the withdrawal data stink like rotting fish on a 40C day. A la the Pfizer vaxxxine study, the treatment groups had a remarkably higher rate of 'withdrawals' than the standard care group, and the researchers don't explain why. This was a randomized trial, and the standard care group was included in the random allocation of subjects, so there should be similar withdrawal rates between groups. Looks to me like the researchers created more 'withdrawals' in the treatment groups to remove subjects with unfavourable data.
Also, zero clinical deterioration and hospitalizations in the combo groups, a mere 9 among the fluvoxamine-only group, yet a whopping 321 of 336 standard care subjects with "mild" 'COVID' experienced clinical deterioration, with many requiring hospitalization?
Yeah, sure.
If this slop was a COVID vaxxxine study, everyone here would be hurling rotten tomatoes at it. Because it's not a vaxxxine, we're supposed to pretend that it's unfairly suppressed research. Sorry, but unblinding, blatant conflicts of interest and extremely suspicious withdrawal and outcome data are not okay just because we're dealing with a non-vaxxxine.
I dismantle this farce of a trial here:
3. The gold standard for drug testing is randomized, double-blind, placebo-controlled trials. Something you don't mention here is that there have already been 5 double-blind trials and 1 single-blind trial of fluvoxamine against 'COVID.' Each and every one showed fluvoxamine to be a dud in the treatment of 'COVID.'
Three of those previous trials were conducted by Reiersen and fellow patent holder Eric Lenze, who has financial ties to Jazz Pharmaceuticals, which now holds the license for fluvoxamine.
In one of their trials, STOP COVID 2, fluvoxamine was such a flop that they didn't even bother publishing the results. The other two failed to show any benefit, so the authors chopped, changed and deleted original endpoints in an attempt to contrive something resembling a significant result. They then tried to word their papers as if fluvoxamine was effective. It wasn't, as I explain here:
Note that the other research groups around the world, who did not have a vested interest in fluvoxamine as a COVID treatment, did not attempt to re-frame their negative results as positive.
Bottom line is that the 3 of 7 trials claiming efficacy for fluvoxamine against COVID just happen to feature Reiersen on the author list, who holds a patent for the use of fluvoxamine against COVID.
4. Fluvoxamine is an SSRI antidepressant. This class of drugs is problematic at the best of times, and fluvoxamine stands out as an especially disagreeable drug. Someone else here wrote that it has a 'superior side effect profile'. Nothing could be further from the truth. It is notorious for causing nausea, agitation, psychiatric disturbances and suicidal behaviour.
The heightened risk of suicidality caused by fluvoxamine is a function of its penchant for causing stimulation and agitation. Depressed people often ideate about suicide, but thankfully most never act upon these thoughts. However, when someone is both depressed and agitated, you have a particularly dangerous state where they are more likely to act upon violent impulses and cause harm to themselves - and others.
The treatment period in the Thai study was 14 days. The peak suicide danger periods with SSRI use occur within the first 4 weeks of treatment, the first 4 weeks after cessation, and after a dosage change.
I've seen with my own eyes what this fluvoxamine junk can do to people, which prompted a 3-part deep dive that you can read at the following links (with plenty of links to the cited studies):
etc. etc.
See, this EMERGENCY virus spawned a lot of people that want to save the world.
“In times when we’re having a global pandemic and there isn’t an approved treatment, if we had a compound that was already available around the world then that might help people sooner. Realistically it’s going to be a while before a vaccine is broadly available.
Dr. Wagstaff, about Ivermectin
This is the same “IT’S AN EMERGENCY” talking point that Steve Kirsch used back in June, 2021 to push SSRI fluvoxamine for *Covid.
Wrote Kirsch:
In a nutshell, the fundamental problem is the medical community is set up to be very careful in approving new treatments. The criteria used, test in a sufficient number of patients to ensure the treatment is both safe and effective, is fine for normal times. But for a pandemic which has killed 500,000 people and for a repurposed drug with a 37 year safety record, the rules should be more accomodating.
There are two problems:
1. The medical community hasn’t really considered how the rules should change in such a situation, so the old rules still apply.
2. The medical community has not seriously looked at all the evidence on the table for fluvoxamine.
They think there are at most two randomized studies. I don’t know of anyone who has looked at all the evidence listed in the FAQ. This evidence was available to everyone on December 1, but nobody other than one reporter (Esther Landhuis) was interested in hearing the evidence (in this case, learning what happened Page 1 at Golden Gate Fields): not the press, not the mainstream medical community.
https://www.skirsch.com/covid/Backstory.pdf
And of course, Dr. Robert Malone makes the same pitch.
It’s an EMERGENCY.
(And we need Pepcid AC and HCQ.)
Press Release: A Multi-site, randomized, Double-Blind, Comparative Trial of the Safety and Efficacy of Hydroxychloroquine and Famotidine for the Treatment of COVID-19 in Hospitalized Adults
When confronting a rapidly moving epidemic or global pandemic caused by a new and highly infectious respiratory virus, time is of the essence, and every day can be measured by thousands more people infected, suffering or dying. The pressing need to rapidly develop drug or vaccine treatments or preventions can be overwhelming. However, those with experience in managing outbreaks are aware of the risks of rushing an unproven drug or vaccine into widespread use before demonstrating both effectiveness and safety; many millions of people could be further damaged if dosed or vaccinated with unsafe medical products. Unfortunately, demonstrating both the safety and the ability to reduce or prevent viral disease for a new drug or vaccine for a new virus takes large amounts of time, money, and risk; usually 10 to 15 or more years, billions of dollars for each candidate, and often ends in failure. The Merck Ebola vaccine was moved through the process from initial human testing to FDA licensure in a record time of five years.
Perfectly reasonable, yes?
We are in an emergency and we need to move rapidly.
This is a Plausible Rationale.
Problem>Solution.
PROBLEM=EMERGENCY
And Tim Sheahan and Ralph Baric and UNC made the same case for Remdesivir.
“This is a game changer for the treatment of patients with COVID-19,” (Ralph) Baric said upon hearing the results of the clinical trial. “Remdesivir provides an effective treatment strategy for the many infected individuals around the globe.”
SPARS also predictively programmed Remdesivir.
From Page 9 of the “fictional” scenario:
At that time, no treatment or vaccine for SPARS was approved for use in humans. The antiviral Kalocivir, which was initially developed as a therapeutic for Severe Acute Respiratory Syndrome (SARS) and Middle East Respiratory Syndrome (MERS), was one of several antiviral drugs authorized in the United States by the FDA to treat a handful of severe SPARS cases under its Expanded Access protocol.
Kalocivir had shown some evidence of efficacy against other coronaviruses, and a small inventory of the drug was already a part of the Strategic National Stockpile (SNS) in anticipation of FDA approval, despite some concerns about potential adverse side effects.
SPARS is…COVID-19.
Kalocivir sounds an awful lot like Remdesivir.
Right about the time the Johns Hopkins planning exercise was scripting the problem and “solution”, UNC was starting to work on just such an antiviral that would come to be dubbed Remdesivir.
From the 2014-2016 SPARS planning exercise script:
At that time, no treatment or vaccine for SPARS was approved for use in humans.
Sounds an awful lot like:
“Right now, there are no approved antiviral drugs for any human coronavirus.”
Tim Sheahan, UNC promoting Remdesivir, 2020
Tim Sheahan, UNC, in the above ☝️ YouTube video published March 19, 2020, Go to :20.
Pay attention to that claim in UNC’s video that Remdesivir worked against all the coronaviruses.
Ivermectin is about to say, HOLD MY BEER, TIM.
Ivermectin is an FDA-approved anti-parasitic drug that has also been shown to be effective in vitro against a broad range of viruses including HIV, Dengue, Influenza and Zika virus.
😮
Can I see all the studies of Ivermectin, the Wonder Drug, working on HIV?
And…Zika?
Is any of this real?
Is this real science?
Although the mechanism by which Ivermectin works on the virus is not known, it is likely, based on its action in other viruses, that it works to stop the virus ‘dampening down’ the host cells’ ability to clear it, Dr Wagstaff said.
We don’t know how it works (in the in vitro cell culture) but work it does!
It dampens down the host cells or something.
(In the Petri dish.)
Hey I’m just going off the spin on this study!
The best for last.
Royal Melbourne Hospital’s Dr Leon Caly, a Senior Medical Scientist at the Victorian Infectious Diseases Reference Laboratory (VIDRL) at the Doherty Institute where the experiments with live coronavirus were conducted, is the study’s first author.
“As the virologist who was part of the team who were first to isolate and share SARS-COV2 outside of China in January 2020, I am excited about the prospect of Ivermectin being used as a potential drug against COVID-19,” Dr Caly said.
Okay, the dude who was the first to ISOLATE THE SARS-COV2 SUPER ANTIGEN OUTSIDE OF CHINA WHERE THE PEOPLE WERE FALLING DOWN DEAD IN THE STREETS…is excited to see how Ivermectin fares at this novel pathogen COVID-19 disease.
How did you isolate the virus?
Okay, that was long, but I’m manic today.
Tomorrow I’m hitting up Conspiracy Sarah again for that Lanka link on how they “isolate” viruses and it is jaw-droppingly ridiculous.
They have quite a process.
It involves calf fetal tissue and like…peanut butter.
Good night, all.
There is mass SHITFUCKERY going on.
I forgot to include the Pierre Kory Fox News DECEMBER, 2020 video clip where he says flat out if you take IVM you will NOT GET COVID.
But this was too long anyway.
Reminded me of Joe Biden saying, "You're not gonna get Covid...if you get vaccinated."
Errybody got a hero and a hero drug. And they all protect the Dangerous Germs EMERGENCY paradigm.
Every last one of them.
"More than 200 strains of viruses that cause colds have been identified, which makes the development of appropriate immunization methods very difficult, if not impossible. For about 40% OF ALL COLDS, the responsible agents have not even been identified."
Biology (2nd Edition) 1989 Raven and Johnson page 576.
So many severe colds in the past... unknown agents... Never identified... Ever... Never...
Okay, given they have trouble identifying causitive viruses normally, there's hundreds of potential viruses to choose from, all NEVER before identified...
Think about this... How did they identify this one SO EARLY??? BY JANUARY 2020????
And determine what they found as the CAUSITIVE AGENT??
THE TIMELINE IS SO SUS...
I can understand they may identify the sequences and virus a few years later, but within days???
Come on...
"People don't ask the right questions"... (Day Tapes)
There's NO QUESTIONING up the chain as to the AUTHENTICITY of this Genebank delivered from CHINA, which was ISOLATED for Genebank SUPER EARLY in Jan 2020...
SO EARLY....
To deliver "known titers" of covid.. with which they'll TEST FOR RELIGIOUSLY using OVER AMPLIFIED PCR. How were these tell tale titers so well known in January 2020?
HOW??
Monash University isolated SARS Cov 2 from one patient only... Why is this so sus?
Two things:
1.) Reverse Transcription Polymerase Chain Reaction (RTPCR) WAS USED, which means that the Melbourne researchers "must have had the information from Chinese authorities" on the selections of primers... Really, they must have had the primers from the Chinese??
Neat trick to get the right primers to detect "covid" so early... What were these primers? The sequences are listed in the research notes, but yet again, thanks CHINA for supplying these sequences which we won't check or verify.
2.) ... Which really leads on from point 1.).... They detected and determined "covid" in Australia on Jan 24, 2020??!!!!
Seriously ONE SICK GUY, a SAMPLE from him yielded the elusive covid...
Wow...
Fortuitously all happened at Monash University ...
Where "Moderna's vaccine production factory will be based at Melbourne's Monash University under a 10-year deal inked with both state and federal governments."... according to government talking points in the media.
So sus that THIS IS THE PLACE to isolate covid in January 2020 isn't it?
Oh and they took a photo... Of a coronavirus...
Hmmmm ...
Could be ANY coronavirus to be honest, one of the known human coronaviruses, or any of the many wild coronaviruses that do not cause human illness... And of course around 40% of viruses that cause colds have never been identified... Never ... So how did they get this one so fast??
https://open.substack.com/pub/geoffpain/p/first-detected-covid19-case-arrived?utm_source=share&utm_medium=android&r=tymb5
In fact it seems 99% of all earthly viruses have never been identified... Because earth is literally swimming in "viruses", every cubic metre of air is something like billions of viruses...
But we isolated this one so quick 🤔... Crapola ...
Not a bad explainer on viruses and just how many of these things are around...
https://youtu.be/Of6BRUWXdUg?si=319hBMd-DeyzvBcP